“Expert” Opinion.
Resorption, Trauma November 13th, 2007
An hour later and this is what I ended up with. I usually don’t charge for lawyer letters, but this one took me away from Heroes so I think I’ll send the lawyer a bill.
As I wrote this I was thinking that the exam candidates could probably quote literature like crazy back at me with respect to dental trauma while here I am slowly forgetting even the classic Andreasen stuff. Oh well, such is life in the fast lane.
Dear Mr. Lawyer,
Thank you for your inquiries regarding XXX (your file No. XXX).
Dental trauma generally leaves involved teeth with a guarded long-term prognosis. This is especially true in luxation injuries (injuries where the tooth is physically displaced in one direction or another and requires repositioning). Delayed sequela of trauma such as internal or external resorption (reduction of root structure), root canal infection, or ankylosis (loss of the tooth’s physiologic attachment apparatus to the jawbone) can become obvious and problematic many years later.
XXX’s tooth 12 currently appears to be in a stable situation and does not demonstrate obvious signs or symptoms associated with infection. This is subsequent to trauma that occurred over three years ago. My testing, however, does provide some evidence consistent with a necrotic (dead) pulp (nerve) within the tooth.
Endodontic (root canal) treatment of a tooth with a necrotic pulp is not essential as a therapeutic measure. Should the pulp become infected, however, endodontic treatment then becomes necessary. The period of time between pulpal necrosis and infection varies from case to case. Generally, the easier it is for bacteria to penetrate into the canal space of the tooth, the faster necrosis will convert to infection.
Because tooth 12 is intact, has no decay, and no pre-existing fillings, the chance of infection developing within the near future is slim. The elective option of pursuing root canal treatment of this tooth for prevention of future potential infection does have some risk associated with it and could actually potentiate other issues with the tooth. I am of the opinion that the tooth should be monitored for problems (through the usual dental recall visits) and any future problems be addressed as needed.
The root canal space within this tooth has become significantly restricted. This is a reactive response of the pulp to the injury before it died off. The more restricted the canal space is, the more complicated root canal therapy becomes and the greater risk of irreparable damage to the tooth whilst searching for an opening into the canal.
At this point the best indicator of retentive potential of the tooth is the external resorption that I observe radiographically. The tip of the root is shorter and more blunted than it should be. This occurred as part of the biologic repair process in the area subsequent to the accident. I have no historic radiographs to compare current ones to, so I am working under the assumption that the resorptive process is currently arrested. If this is not the case and the resorption is in fact progressive, the tooth will be lost once the root is completely resorbed. This is the other reason the tooth needs to be monitored for some time longer at regular recall intervals.
Should this tooth be lost, an osseointegrated dental implant would be the ideal way to replace it. Permanent tooth replacement is best done once jaw growth is complete (generally between 18 and 21 years). Because my expertise lies within the bounds of retaining this tooth, replacement options, costs, and timeline are best discussed with the dentist who would actually perform the replacement procedures.
I hope this information is of value to you. Should you wish to further discuss this case, please feel free to email me at XXX.
Sincerely,
Ameloblast
Is Nothing Up?
Resorption, Trauma November 13th, 2007
Hmm. A blank window to post in—and nothing to post. Nothing interesting anyway.
Am I too busy, not busy enough, was life more interesting a few months ago when I was posting like mad to the blog?
It’s a combination of all of the above, but most importantly, it’s because of Facebook.
I’ve recognised my problem, admitted it, now I have to change. I’ll waste less time on Facebook, and waste more time posting to the blog. Now I only have to find things to post about…
I’ll think about that once I finish this dento-legal letter to a lawyer asking for a translation to lay terms of a case about a boy who suffered an intrusive luxation injury a few years ago and presented to me for exam a couple of months ago. The lawyer needs to know what the terms ankylosis, calcific canal obliteration, and apical external resorption mean.
Internal Resorption Update.
Resorption July 26th, 2007
Here’s a month and a half recall film of the internal resorption case I did a short time ago. I just finished a retreatment of 11. Looks like healing is progressing apical to 21. The tooth has remained comfortable.

Crazy Internal Resorption.
Resorption June 6th, 2007
Internal resorption cases that are as advanced as this are difficult to manage, especially if they are more coronal and compromise the integrity of the clinical crown.
The complication with this case was that the resorption had perforated the root. Every time I tried to regain the true canal beyond the level of the defect, my instruments would slip out the perforation. Because of this I decided to limit obturation to the level of the defect.
The prognosis of the case is fair at best and could require surgery to address the apical area and perforation better. Surgery isn’t a great option either though because if I end up having to resect the root to the coronal aspect of the defect, the tooth might become very unstable.
Ankylosis.
Resorption, Trauma January 16th, 2007
This guy was asymptomatic. A removable partial denture hooks onto the 13 and the endo in 12 was done 1.5-2 years ago. His current dentist was a little concerned with the resorptive defect on the root of the 3.
Percussion produced a higher pitched sound on the 3 than the other teeth, and when I checked mobility, there wasn’t even any physiologic movement.
I diagnosed ankylosis, replacement/external resorption, and told the patient to consider replacement options. He’ll probably be ok leaving the tooth alone for now, but if the replacement resorption is still active, he will lose the tooth sooner than later.
These are all things we see with teeth that have been traumatised (avulsed in particular). The only history of “trauma” to this tooth is the RPD…
